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This enhancing effect on glucagon secretion makes lone GIP agonists unsuitable for the treatment of T2DM [23]
Similar immune defects are observed in alcohol-related cirrhosis, including depletion and functional impairment of MAIT (mucosal-associated invariant T) cells and elevated soluble immune checkpoint molecules, such as sTIM-3 (62)
JCO Clin
(2022) suggests that GLP-1 RAs may be associated with a reduced risk of atrial arrhythmias
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