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These microorganisms, through Various direct and indirect pathways can trigger neuroinflammation, promote A(A) accumulation, and impair neuronal function, key processes involved in the pathogenesis of AD
GLP-1 receptors are expressed in the atrial myocardium and the sinoatrial node
As mentioned above, CSE-induced cell death in rat VSMCs can be completely inhibited by specific ferroptosis inhibitors and an iron chelator
Both approaches complement each other
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