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Therefore, it would be crucial to understand and outline the specific contribution of each cell type MN, glia, or others to ALS, in order to strategically target them with the NRF2 activators, thereby restoring neuronal homeostasis and survival, and functional MN-glia interactions (Fig

Current research has validated the regulatory effects of certain clinical medications on the Th17/Treg balance, as summarized in Table 1

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