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Moreover, endogenous DNA, originating from nuclear damage or immunogenic cell death (ICD), has been demonstrated to activate cytoplasmic PRRs such as cGAS and AIM2
Improvement of metabolic control and quality of life
These findings identify ALC as a metabolic regulator that restores social isolation-induced myelin deficits by coordinating myelin protein synthesis, mitochondrial function, and myelin lipid remodeling
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