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Our results suggest that both increased S -glutathionylation processes and suppressed de-glutathionylation reactions contribute to hepatic S -glutathionylation in NAFLD
This review examines the intricate relationship between cigarette smoking, smoking cessation therapies and anti-TB drugs, focusing on the impact of tobacco smoking compounds on liver detoxifying systems, such as influence of polycyclic aromatic hydrocarbons (PAHs) on hepatic cytochrome P450 (CYP450) enzymes mostly, and on metabolism of antituberculous medications
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