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The vitamin is then transferred to TC2, which serves as the circulating plasma transporter, hereditary defects in the production of TC2 and its receptor may produce functional deficiencies in B 12 and infantile megaloblastic anemia, and abnormal B 12 related biochemistry, even in some cases with normal blood B 12 levels

It has been investigated for its potential involvement in regulating sleep patterns, neuroendocrine function, and stress-related hormone signaling

The study examined safety parameters and metabolic biomarkers over a 12-week treatment period [2]