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Targeting glutamine metabolism with a novel Na+/K+-ATPase inhibitor RX108 in hepatocellular carcinoma
Eur J Endocrinol (2012) 166(2):24753
The No-DAC version more closely mimics natural pulsatile GH release patterns and is generally preferred for this reason
3 EMT functions as a reversible cell-state program that endows tumor cells with increased motility, survival capacity, and tolerance to therapeutic stress, thereby contributing to invasion, metastasis, and treatment resistance
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