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Over-expression of miR-17-92 in HUVECs can protect ECs from erastin-induced ferroptosis by targeting the zinc lipoprotein (A20)-acyl-CoA synthetase long-chain family member 4 (ACSL4) axis that could protect glutathione peroxidase 4 (GPX4) and inhibit ROS production (Xiao et al., 2019)
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Reduced availability of NO occurs when there is a decrease in the expression or activity of endothelial NO synthase (eNOS), actively participating in the atherogenic process
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