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The primary mechanism observed in animal studies involves the upregulation of vascular endothelial growth factor receptor 2 (VEGFR2) and the activation of focal adhesion kinase (FAK) and paxillin pathways
The nuclear factor erythroid 2-related factor 2 (Nrf2)/antioxidant response element (ARE) signaling pathway is an important mediator of cellular response during an oxidative stress condition (Zhang S
10.1007/s10815-014-0390-1 119
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