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656 Moreover, the AAV2/8 vector can mediate the expression of the human ETHE1 gene in the liver of ethylmalonic encephalopathy mouse models, effectively clearing circulating hydrogen sulfide, correcting plasma thiosulfate levels, restoring sulfur dioxygenase activity, significantly improving disease phenotypes, and extending lifespanhighlighting its potential for future clinical applications in treating ethylmalonic encephalopathy

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In the absence of a proper code, the claim may not be covered by medical necessity and is likely to be rejected

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