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These cells, which accumulate with age, no longer divide but resist programmed cell death, contributing to inflammation and tissue dysfunction
It has also been demonstrated that inhibitory GABAergic CNS GIPR + neurons are necessary for the food intake reducing effects of GIPR agonists and dual incretin co-agonists (54, 55), while excitatory glutamatergic CNS GLP-1R + neurons are necessary for the food intake reducing effects of GLP-1R agonists (71)
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And these drugs are opening up opportunities for a once-small, but now growing, category of RTD shakes
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