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(2020) specifically addressed NSAID-induced gut damage a clinically relevant injury model and documented that BPC-157 administration stabilized the claudin-4 and occludin tight junction proteins that are degraded by NSAID exposure, providing a mechanistic explanation for the peptides permeability-protective effects
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The commodity peptides stayed cheap and stayed put
BPC-157 requires robust safety trials in humans first, then trials to determine the dose-response required to elicit meaningful therapeutic regenerative changes, followed by trials to establish the long term safety
And it turns out to be a fragment a small piece snipped off a much larger protein [3]
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