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BDNF-Adjacent Pathways: While GHK-Cu does not directly upregulate BDNF (Brain-Derived Neurotrophic Factor), its anti-inflammatory effects in the CNS specifically its reduction of microglial activation may create conditions conducive to BDNF signaling
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BPC-157s apparent ability to modulate MMP activity suggests a role in preventing excessive collagen degradation, thereby supporting more effective matrix remodeling and improving the overall quality of repaired soft tissue
