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Ferroptosis and AD pathology Amyloid (A) plaques and neurofibrillary tangles (NFTs) A plaque accumulation is a hallmark pathological feature of AD, where abnormal A aggregation disrupts synaptic function and impairs memory (30,31)

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Individuals with genetic variants in IL6, IL6R Targeting the kynurenine pathway: Niacin, magnesium: In addition to responding to inflammation or stress, the body also shunts more tryptophan towards the kynurenine pathway when niacin is low.[ref] Additionally, magnesium is needed to convert quinolinic acid into niacin

Duncan Macewan Person And you can see those adjustments over time

By selectively disrupting key antioxidant systemssuch as inhibiting glutathione synthesis with buthionine sulfoximine (BSO), blocking thioredoxin reductase with auranofin, or directly targeting NRF2therapies can push cancer cells beyond their limited oxidative buffering capacity