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Studies show improved outcomes in models of osteoarthritis and joint damage, likely mediated through growth factor upregulation and anti-inflammatory pathways
Modulation of CYP51A1 The expression and activity of CYP51A1 are tightly regulated through both transcriptional mechanisms and post-translational modifications (PTMs), including ubiquitin-mediated degradation and proteinprotein interactions (PPI) (Fig
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AHK-Cus direct follicular effects (DPC proliferation, VEGF upregulation) and GHK-Cus broader regenerative gene modulation (collagen synthesis, matrix remodeling, anti-inflammatory effects) address different aspects of scalp and follicle biology