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Consequently, despite the potential for higher initial blood levels, the inherent physiological immaturity and reduced somatic mass associated with preterm birth mean that these infants have significantly diminished endogenous L-carnitine reserves
, 47- ( )
Carnitine then binds to VLCFAs to form the corresponding acylcarnitines, allowing them to leave the peroxisome and eventually enter the mitochondria for complete -oxidation [29]
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