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, 48 , 303-307, doi: Dequanter, D., Van De Velde, M., Bar, I., Nuyens, V., Rousseau, A., Nagy, N., Vanhamme, L., Vanhaeverbeek, M., Brohe, D., Delre, P., Boudjeltia, K., Lothaire, P., and Uzureau, P
The process also creates harmful compounds that bond to and drain existing stores
Semax, an analog of ACTH(4-10) with cognitive effects, regulates BDNF and trkB expression in the rat hippocampus
Because typical early atherosclerotic lesion formation observed in hyperhomocysteinemic animals can be prevented by an antiplatelet therapy with dipyridamole, the authors concluded that platelet-mediated proliferation of smooth muscle cells is a potential cellular mechanism involved in the atherogenicity of hyperhomocysteinemia
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