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Robert Kushner: GLP-1 appears to be a strong enough regulator, dealing with the appetite dysregulation, to really harmonize my understanding harmonize all these other factors you're talking about
This participant was already taking tirzepatide when she enrolled in the trial, but before any stimulation was delivered, giving us a unique opportunity to make foundational observations about how the drug alters brain signals. At first, results were promising: As patient 3s tirzepatide levels reached full dosage, craving-linked NAc activity in her brain went dormant and she had no obsessive thoughts about food
These agents may stabilize metabolic risk factors, but nerve regeneration has not been conclusively proven
Apr 4 2023;13(4)doi:10.3390/brainsci13040613
