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On the one hand, the pathophysiological mechanisms underlying SIM are complex, involving processes such as inflammation, oxidative stress and metabolic dysregulation, which pose challenges to the development of targeted treatments
Sounds good in theory
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Given the reabsorption of renin by proximal tubule cells through megalin, and the broad expression of ACE within the kidney, allowing locally synthetized ANG II independent action, the interference of SGLT-2-i with intrarenal RAS could explain such results
S., & DeLany, J
