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Exendin-4 Attenuates Remodeling in the Remote Myocardium of Rats After an Acute Myocardial Infarction by Activating -Arrestin-2, Protein Phosphatase 2A, and Glycogen Synthase Kinase-3 and Inhibiting -Catenin
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10.1016/j.toxicon.2006.09.012 119 HerringtonJ.SanchezM.WunderlerD.YanL.BugianesiR
These mechanisms are investigated to better understand how multi-receptor agonist peptides influence intracellular signaling cascades, receptor binding dynamics, and downstream molecular responses
The mechanism by which TMAO causes atherosclerosis will help us design appropriate drugs for optimum cardioprotection and better guide the diet of patients with myocardial infarction or coronary heart disease
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