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MOTS-c is a mitochondrial derived peptide studied for endurance and metabolic recovery

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Our findings revealed that after transfection with STAT3-targeting siRNA (siSTAT3), the stimulant effect of GHK-Cu on the healing of MCECs and the effect on the protein expression of ZO-1 and Occludin is canceled

PMID: 6548970

The precise mechanisms of drug resistance in tumors are complex and multifactorial, but they can be grouped into three categories: insufficiency of pharmacokinetic properties, intrinsic factors of tumor cells, and external conditions of tumor cells in the tumor microenvironment (TME) [3]