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The co-crystal structure of Compound 71 bound to PDE10A revealed that, in addition to existing interactions, an extra halogen bond formed between the chlorine atom of Compound 71 and Y524, contributing to enhanced inhibitory activity (Figure 18B)

12 Moreover, lipotoxicity, endotoxin translocation, and chronic low-grade inflammation activate Kupffer cells (KCs) and polarize immune cell populations, 13 contributing to a persistent pro-inflammatory hepatic microenvironment

For that reason, research-only context matters

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Either way, the most important thing is that you are addressing nutritional gaps during GLP-1 therapy