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Possibly, the loss of particular TJ proteins such as occludin is a result of increased proteasome-mediated degradation observed in IBS triggered by low-grade inflammation and resulting in increased intestinal permeability [196]

Technically, its a synthetic peptide fragment derived from a protein in the human gut

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It shortens the telogen (resting) phase, prompting dormant follicles to enter the anagen (growth) phase prematurely