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the remaining seven forms are uncharacterized in humans Bioavailability remains suboptimal, with oral administration largely ineffective due to enzymatic degradation
doi:10.1016/j.arcmed.2019.04.004
Additionally, O 2 can react with nitric oxide (NO) to generate excessive ONOO, activating caspases, causing lipid peroxidation, and leading to either apoptosis or necrosis [49]
Consistent with the pharmacological results, the above results provide convincing evidence that the GPX4 overexpression protects TECs from ferroptosis and mitigates renal interstitial fibrosis
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