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doi: 10.1111/j.1365-3164.2006.00551.x
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Beyond small clinical cohorts, large-scale patient outcome data strongly supports the use of these compounds
21,22 Another suggested hypothesis is that ethanol may directly enhance the activity of NAD(P)H:quinone reductase, serving to reconvert quinone metabolites back to native APAP and thereby limiting the accumulation of toxic metabolites
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