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The connection between thyroid function and intestinal microbiota has been suggested in animal models since the 1970s, and intestinal dysbiosis has been reported to influence iodide absorption and enterohepatic circulation from the intestinal lumen, both of which have an impact on thyroid hormone amounts

Although subcutaneous insulin administration could cause the enhanced CETP activity in the latter example [34], our results suggest that the lower level of CETP-inhibiting ApoC1, observed in the ND individuals, may also play a role