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Accessed May 21, 2025.
As there is no effective therapy to treat CV injury and fibrosis associated with T2DM, DPP-4 inhibitor-mediated targeting of SDF-1, to prevent its degradation and enhance stem cell mobilization from bone marrow and recruitment to peripheral tissue, may be an attractive strategy to enhance CV tissue repair [35, 38, 197,198,199]
This becomes relevant for diabetes because blocking the SGLT2, and therefore preventing that reabsorption, allows excess glucose to be excreted in the urine rather than returned to the bloodstream
Any frequent or unexpected changes in blood glucose levels should be reviewed by a clinician to ensure treatment remains safe and effective
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