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During mealtime, the network presented in this study driven by OB GLP-1R may contribute to a decrease in SNS activity, and thus participate in the increase of glucose-induced insulin secretion, in concert with systemic GLP-1 and the GLP-1 dependent neuronal circuitry of the brainstem and hypothalamus recruited under normal physiological circumstances

Jonathan Hommel for guidance in the original study design, and Dr

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The same shuttling system can also export excess acetylcarnitine out of the mitochondria to limit inhibition of pyruvate dehydrogenase and spare the pool of free CoA necessary for other molecules

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