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2011), and enhancing the endogenous antioxidant capacity, including the upregulation of CAT, SOD, and GSH-Px activities (Augustyniak and Skrzydlewska 2010
The mechanism likely involves both the anti-inflammatory properties of BPC-157 and its promotion of collateral blood flow to oxygen-starved tissue
[14] As a result, it has been described as over-prescribed for this application
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