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Although our data also indicate that, following chronic LH GLP-1R silencing, the brain attempts to compensate for the reduced anorexic signal in the LH by potently increasing GLP-1R expression in the NAc only in males and preproglucagon in the NTS of both sexes

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Such precision medicine strategies could substantially enhance GHK-Cu's therapeutic value while supporting more efficient clinical trial designs in future development programs, approaches increasingly employed across peptide therapeutic development for diverse clinical applications