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Collectively, these data support the conclusion that increased cellular GSSG accumulation in hepatocytes plays a critical role in the pathogenesis of NAFLD and may represent a potential therapeutic target for the treatment of NAFLD
Two practical consequences fall out of this mechanism, and both explain the shape of the protocol below
Genotype/phenotype relation As new genetic variants related to PFIC were discovered, genotypephenotype relationships emerged [19]
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