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Gillingham M, Van Calcar S, Ney D, Wolff J, Harding C: Dietary management of long-chain 3-hydroxyacyl-CoA dehydrogenase deficiency (LCHADD)
[1] At the cellular level, the tendon work is genuinely mechanistic: in cultured rat tendon fibroblasts, BPC-157 accelerated cell outgrowth, improved survival under oxidative stress, and increased migration in a dose-dependent way, apparently through the FAKpaxillin pathway
Research literature also describes TB-500 as influencing inflammatory signaling pathways and cellular responses associated with tissue remodeling
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Its most prominent function has been suggested to regulate apoptosis in senescent cells
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