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This phenomenon is often exploited in cancer treatments, where therapies aim to increase ROS levels beyond a threshold that cancer cells can tolerate, effectively inducing their death while sparing normal cells [9]
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We observed a drastic decrease in cell viability especially after transduction with sgRNF4_3 in both cell lines, whereas transduction with sgRNF4_1 affected viability only in MV4-11 cells ( Figure 1C, D )
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