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Modulation of enzymes involved in NAD + consumption and synthesis In addition to NAD + precursor-based strategies, halting of processes linked to NAD + consumption or biosynthesis represents a complementary and therapeutically appealing approach to alter or restore NAD + homeostasis in context-specific applications
X(PC)-H N induced potent cell death in SUP-B15 cancer cells, while catalytically inactive X(PC)-H N and X(PC) protease alone had no effect (Fig
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