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The original Achilles tendon transection model in rats (Staresinic, Sikiric et al., 2003) showed that systemic BPC-157 accelerated healing of fully transected tendons, and this finding has been reproduced across multiple labs over the 20+ years since
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According to preclinical research published in peer-reviewed journals, BPC-157 has been studied for its potential interactions with several biological pathways in animal models: VEGF (Vascular Endothelial Growth Factor) Pathway: Animal studies suggest BPC-157 may interact with angiogenesis pathways
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