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The importance of oxidative stress in asthma is further highlighted by evidence from studies that proved oxidative stress can alter immune responses, such as the Th1/Th2 balance, and activate pro-inflammatory pathways such as NFB, which exacerbate airway inflammation [125, 129]
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Although these data suggest that GSH depletion per se, at least for a short period of time, is not sufficient to cause cell injury, a prolonged depletion of mucosal GSH leads to severe degeneration of intestinal mucosal epithelial cells, as shown in BSO-treated mice (Martensson et al, 1990)
Arthritis Rheuma (1998) 41(11):198794
