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doi: 10.3390/ijms26157080 14
Although GIP receptor activation may exert anti-emetic effects through brainstem modulation, these effects appear insufficient to completely counterbalance GLP-1 mediated emesis during early treatment and dose escalation ( In contrast, glucagon receptor activation is independently associated with nausea and vomiting through both central and peripheral mechanisms, including effects on gastric motility and autonomic signaling
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There were more lipid droplets and inflammatory cell infiltration in hepatocyte cells
see above), and total glutathione (and Asc) level increased markedly
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