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Krjk ezt figyelembe vve a termket els felbontskor helyezze 1-2 rra htszekrnybe, s felrzs nlkl, vatosan nyissa ki
ITPR2-dependent ER-to-mitochondria Ca2+ transfer has been shown to promote senescence, whereas Itpr2 deficiency reduces ER-mitochondria contacts and is associated with attenuated aging phenotypes in mice, supporting a role for contact-dependent Ca2+ signaling in age-related dysfunction (234)
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clinical practice has shown it delays gastric emptying, increases satiety, and reduces appetite
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