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These findings suggest that pharmacological inhibition of FSP1 may provide an effective strategy for cancer therapy by sensitizing cancer cells to ferroptosis-inducing chemotherapeutic drugs and, if used in combination with GPX4 inhibitors, could have a strong synergistic effect

Vitamin C regenerates oxidized glutathione back into its active form, creating a self-reinforcing brightening cycle

Glioma-induced SIRT1-dependent activation of hMOF histone H4 lysine 16 acetyltransferase in microglia promotes a tumor supporting phenotype

LDHA promotes osteoblast differentiation through histone lactylation