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Tumor risk via p53 pathway interference Mitigate risk by: Careful patient selection: exclude patients with pre-existing active malignancy or high tumour risk (e.g., known p53 mutations, familial cancer syndromes)
These components act as vigilant cellular guardians, protecting against damage caused by free radicals
Tan CL, et al
Safety risks primarily arise from unverified sources and non-compliant injectable routes
Sustained IGF-I elevation from any GH secretagogue raises theoretical concerns IGF-I is a mitogen, and chronic elevation is a variable of interest in tissue-growth contexts that have not been evaluated for this blend over time
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