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Ischemia has been with a metabolic shift away from -oxidation to the use of glucose of a substrate
10.1089/ars.2007.1528 Antioxid
Knowing what to expect based on the clinical trial literature helps distinguish between expected effects and anomalies that warrant investigation
Activation of GLP-1R slows gastric emptying, enhances glucose-dependent insulin secretion from pancreatic beta cells, suppresses inappropriate glucagon secretion, and produces a robust central satiety signal via vagal afferents and hypothalamic circuits
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