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The importance of targeting the underlying pathophysiology of T2DM rather than focusing management on reducing plasma glucose levels, as is currently recommended, has recently been debated [8]
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Microglial TNF--dependent elevation of MHC class I expression on brain endothelium induced by amyloid-beta promotes T cell transendothelial migration
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The retro-inverso D-amino acid configuration of FOXO4-DRI confers resistance to proteolytic enzymes, a pharmacokinetic property critical for maintaining peptide stability in complex biological research environments
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