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Thus, the presence of the basic residue seems to be a necessary but not a sufficient condition for GSH activation

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The review also documented how GCL expression is transcriptionally upregulated by electrophiles through antioxidant response elements (ARE/EpRE) in the GCLC and GCLM promoters establishing the Nrf2/ARE pathway as the master regulator of GSH biosynthetic capacity under stress conditions

It seems that GSH sacrifices itself to regenerate other antioxidants and a large amount of GSH is consumed as a direct antioxidant and also in recycling other antioxidants