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These findings provide a solid mechanistic foundation for the future development of BPC157-based or sequential therapeutic agents targeted at enhancing vascular repair

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Its main function is to increase energy expenditure by inducing cold- or diet-stimulated heat production (van der Lans et al., 2013), and by uncoupling oxidative phosphorylation from ATP synthesis through the uncoupling protein-1 in BAT mitochondria (Cannon and Nedergaard, 2004

X(PC), which cleaves procaspase-1 between Glu 314 and Asp 315, displayed strict specificity at positions P3, P2, P1, P1, P2 and P3 (Fig