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Anti-hyperlipidemic effects of GLP-1RAs In individuals with T2DM, lipid abnormalities are vital contributors to the risk of ASCVD, including the risk of ischemic stroke [35]
That same receptor is the target GLP-1 medications are designed to engage
Research context Each component has independent receptor-pharmacology literature
Human eNOS+ enteric neurons in the submucosa also express the GLP-2R, supporting a direct role for GLP-2-mediated increase in eNOS protein and NOS release through cAMP-dependent protein kinase A (Table 3)
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