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Mechanistically it works through the Folate-AICAR-AMPK pathway, disrupting the folate-methionine cycle in a way that mirrors some effects of methotrexate, without the same toxicity profile (Wan 2023)
Together, our results suggest the presence of an aging-dependent increased pool of progenitor cells, which could be compatible with impaired differentiation into effector cells
These symptoms usually resolve quickly and arent considered serious
Identify hepatotoxic effects of medications using available resources
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