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Moreover, in vitro and in vivo models indicate that the activation of PPAR can inhibit HCC cell proliferation and tumor growth by inducing cell cycle arrest and apoptosis by means of the regulation of a panel of downstream effector molecules, such as p18, p21, p27, p53, and caspase-3/7 and caspase-8, respectively (Figure 4)

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9.2 GSH-triggered drug release and prodrug activation strategies GSH-responsive drug delivery and prodrug activation systems often exploit its high affinity for metal ions to trigger selective drug release within the tumor microenvironment (TME)

SEC: size exclusion chromatography

If you experience significant side effects at any dose, you can remain at that dose for 6-8 weeks before escalating, or temporarily step back to the previous dose