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EOFAD is characterized by early onset, family history, a variety of neurological symptoms, and a more aggressive course compared to sporadic AD (Wu et al
Strategies to enhance the tolerability of GLP-1-based therapy might include identification of agents that stimulate release of endogenous GLP-1 from intestinal L-cells, potentially accompanied by fewer adverse effects
Clinically, this localized oxidative damage in the brain translates into the severe cognitive dysfunction, memory impairment, sensory overload, and profound "brain fog" that so many patients with chronic neuroimmune conditions endure daily
Nature 444 , 840846 (2006)
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