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The mechanism involves upregulation of growth hormone receptors in tendon fibroblasts and increased collagen synthesis at the repair site
Wright AK, Carr MJ, Kontopantelis E, Leelarathna L, Thabit H, Emsley R, et al
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Another important factor was it had the capacity to bind to albumin, a large protein common in the bloodstream, which would make the GLP-1 molecule too large to be quickly cleared by the kidney
