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Lower susceptibility of female mice to acetaminophen hepatotoxicity: role of mitochondrial glutathione, oxidant stress and c-jun N-terminal kinase
Semidehydroascorbate is reconverted to ascorbate in the cytosol by cytochrome b 5 reductase and thioredoxin reductase in reactions involving NADH and NADPH, respectively
To further characterize these interactions, we constructed a molecular interaction network (Figure 7C) revealed three functionally interconnected modules: ( Consistent with multi-omics findings, KEGG pathway analysis identified glutathione metabolism as the most significantly perturbed shared pathway (Figure 8A), exhibiting coordinated changes between upregulated antioxidant enzymes such as Gpx3 and Mgst1 alongside depleted glutathione derivatives, supporting redox imbalance as a key pathogenic feature
The Role of Liposomal Delivery To overcome the absorption barrier, we utilize liposomal delivery
